What are three alternative fates of a new gene duplicate? What is the evidence that gene duplication has been the source of the α and β gene families for human hemoglobin? DNA-sequencing studies for a gene in two closely related species produce the following numbers of sites that vary:
Synonymous polymorphisms 50
Nonsynonymous polymorphisms 20
Synonymous species differences 18
Nonsynonymous species differences 2
Does this result support neutral evolution of the gene? Does it support an adaptive replacement of aminoacids? What explanation would you offer for the observations?
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